Early Detection of Alzheimer Disease—A Virtual Town Hall Meeting
health issue, and if so, is there a national plan to address it (Brookmeyer et al., 1998)? Q: For Drs.
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Not Dead Yet: Estrogen Deserves Another Chance
itself and has brain-specific aromatase (see Sasano et al., 1998). Dominique Toran-Allerand Regarding
Grasping the Shadow Force: Immune Cells in ALS
related news story on Lincecum et al., 2010). Compounds that block the interaction between CD40 and its
Alzheimer's: A Triple Whammy. Why are So Many Neurodegenerative Diseases Single, Double, or Triple Amyloidoses?
concept of synaptic apoptosis developed by Mattson et al., 1998 John Q. Trojanowski One would assume
Suzanne Hendrix on Constructing Composites for Trials in Early Alzheimer’s
Have you ever been jolted wide awake by—biostatistics? That’s what happened on the last morning of the Clinical Trials on Alzheimer’s Disease conference held November 5-7 in Barcelona, Spain. At 8 a.m., as sleepy CTADeers slowly shuffled in, Suzanne Hendr
Leaky Blood-Brain Barrier a Harbinger of Alzheimer's?
Cullen et al., 2005; Goos et al., 2009; and Feb 2014 news). Iron, possibly from the breakdown of
Is Alzheimer’s Disease a Uniquely Human Disorder?
e.g., Bussiere et al., 2003). Brain regions giving rise to long axonal connections have expanded in
Computational Modeling—Will it Rescue AD Clinical Trials?
simulation tool (CTS) (see Jul 2013 news and Rogers et al., 2012). Klaus Romero and colleagues at CAMD
Mutations Impair TREM2 Maturation, Processing, and Microglial Phagocytosis
In the July 2 Science Translational Medicine, an international group of scientists reported how TREM2 missense mutations linked to neurodegenerative diseases impair the protein's function. The mutations prevent TREM2 from reaching the cellular surfac
Deficiency of Presenilin 1 Inhibits the Normal Cleavage of the Transmembrane Domain of Amyloid Precursor Protein
report by De Strooper et. al., (1),and the accompanying commentary by Haas and Selkoe (2), conclude that
Isolation of Single Immunohistochemically Identified Whole Neuronal Cell Bodies from Post-Mortem Human Brain for Simultaneous Anlaysis of Multiple Gene Expression
et al., (Int. J. Neurosci., 1990, 55:81-88.). Question 3) What percentage of the brains that were
Alzheimer Presenilins in the Nuclear Membrane, Interphase Kinetochores, and Centrosomes Suggest a Role in Chromosome Segregation
IL-1-positive microglia in DS brain of all ages (Griffin et al, 1989). Mqxia: Hi, nice to be back here. June
Frameshift Mutants of β Amyloid Precursor Protein and Ubiquitin-B in Alzheimer's and Down's Syndrome Patients
aberrant splicing events exclusively in sporadic ALS patients (Lin et al., 1998). The splice variants were
Apoptosis in Alzheimer Disease
ultimate cell death. Again, I refer you to our data (Mattson et al., Exp. Neurol. 153:35-48 and Mattson et
Pin1, Tau and the Cell Cycle in Alzheimer Disease
mitosis[ref.5]). Summary of Paper Under Discussion In the paper being presently discussed, Lu et al. provide data
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